1. Volume repletion most patients 3-6L down. When sugars < 250 include dextrose containing fluids.
2. Electrolyte repletion K<3.5 consider stopping insulin, K3.5 to 5.5, consider adding K to fluids 20-30meq/L. <5.5 no need to add potassium. Check Mg, Phos levels as well.
Insulin drip >1units/kg dose. Switch to subQ after gap normalized and bicarbonate normalized.
Hyperosmolar hyperglycemic state
Triad
Severe hyperglycemia (> 600 usually)
Elevated serum osmolality (>320 osm/kg)
Altered mental status
Treat similarly to DKA, usually require more fluids as patients more dehydrated.
Resuscitative Hysterotomy (Ben Turner and Harrison Brown)
Indications to do
Maternal cardiac arrest without ROSC within 4 minutes
Estimated gestational age of infant >20 weeks (fundus > 20 cm)
Physical with pelvic with possible cervical motion tenderness Lower abdominal tendernes, Uterine and or adnexal tenderness, Mucopurulent discharge, Fever
DX: Sexually active women with lower abdominal pain without other cause found with PE with CMT, Adnexal tenderness, or uterine tenderness
Tubo-ovarian Abscess
Dx via ct or US
Needs admission for IV antibiotics and OB/GYN Consult. Can have IR drainage.
Predictors of failure of antibiotic treatment: WBC 16k or abscess > 5.2 cm
Outpatient management
Rocephin 250mg IM + Doxycyclin 100mg Po BID x 14 days with Metronidazole 500mg Po BID x 14 days
Dispo
Admit of they have TOA, pregnant, cannot tolerate po, septic, failed outpatient antibiotics
Ovarian Torsion (Tyler Bayers)
Ovary can rotate around suspensory ligaments or utero-ovarian ligament, compression of ovarian vein with leads to obstructed venous outflow leading to ischemia and necrosis
Risk factors mass >5cm, hx of cyst, ovarian malignancy, TOA, pregnancy
Presentation
90% with pelvic pain, Adnexam mass, nausea and vomiting
Pearl- Right sided torsion more common due to presence of sigmoid colon on left
Ultrasound- Evaluate for decreased venous/arterial flow
2/3rd might have normal flow
Can see enlarged unilateral ovarian volume
Lpelvic free fluid
Loss of echogenicitiy
Whirlpool sign
CT
Can be used for evaluation of suspected torsoin,
Sensitive for secondary findings in torsion
Can find ovarian enlargement, ovarian mass, distended pedical, lack of enhancement
Management
Pain and nausea control, transabdominal/transvaginal US, Emergent OBGYN consult
Definitive diagnosis by direct visualization
Bartholin Gland Cyst/Abscess (Dan Fischer)
Ducts of the glands drain into posterior vestibule at 4 oclock and 8oclock positions. See mass near the posterior introitus medial to labia minora
Usually sterile initially but can become infected
Abscess- erythema, fluctuance, severely painful
Dx- Clinical consider sti testing
Tx: Word catheter placement
Make small incision so that word catheter will not fall out
Drain abscess, explore wound
Place word catheter and inflate balloon with 2-4cc of water with blunt kneedle
Keep in place for 4-6 weeks
Fllow up with gyn
HTN emergencies in Pregnancy (Joshua Sennets)
Can occur from 20 weeks gestation to 6 weeks post partum
Definition- new onset htn and proteinuria
With severe features- new onset htn and signs of end organ dysnfucntion after 20 weeks gestation and up to 6 weeks gestation
A 26-year-old G1P0 38 weeks and 4 days female presents as a Code Green to the ambulance circle. The patient brought back to Room 9 for assessment. The patient states her “water broke” and it soaked through her pants. She is feeling contractions. Vitals are HR 120 BP 128/78 RR 24 SPO2 100% and T 97.6 F. On external vaginal exam you palpate a pulsating vessel in the vaginal canal and fetal head at 0 station.
● What is the diagnosis? Umbilical cord prolapse
● What is the most important next step in the management of this patient? Elevate presenting part to reduce compression and transport to OR for emergent c-section
● What positions can you place the patient in to alleviate pressure on the protruding part? Knee to chest position, no pushing or Valsalva
A 27-year-old G1P0 at 37 weeks and 4 days is presenting in active labor to the emergency department. She is endorsing painful contractions that began within the last hour. Contractions are occurring every 2 minutes. Vitals are HR 106 BP 136/68 RR 18 SPO2 98% and T 98.8 F. Physical exam reveals fetal head crowning and bloody show. During delivery, you are able to advance the posterior shoulder, however as you attempt to advance the posterior shoulder the fetal head retracts. An episiotomy is made; however, you are still unable to advance the anterior shoulder.
● What is the diagnosis? Shoulder dystocia
● What is the most appropriate next course of action?HELPERR pneumonic. Chall for Help, Evaluate for episiotomy, Legs flex (Mcroberts maneuver), Pressure (suprapubic pressure), Entry maneuvers (Wood’s corkscrew or Rubin II maneuver, Remove posterior arm by sweaping it across chest, R Roll on all fours
● What are some risk factors for this condition?Preterm labor, macrosomia, small materanal pelvis, prolonged labor
● Failure to recognize this condition and correct it can have what damaging results? Fetal demise
● Which of these three maneuvers is the most effective in relieving this condition? Roll on all four
Case 3:
A 21 year-old G4P3 woman presents to your Rural Emergency Department in Ashland, KY in active labor and has a spontaneous vaginal delivery in the emergency department. Prenatal care was appropriate and the patient’s blood type is O-positive. The infant is full-term and well. An intact placenta passes shortly thereafter, followed by vaginal bleeding. There are no obvious lacerations to repair, and the bleeding appears to be originating from the cervical os. Vital signs are within normal limits. The patient continues to have oozing of blood from the vagina several minutes after birth.
What is the diagnosis? Post partum hemorrage
What is the most common cause of this diagnosis? Uterine atony
What is your next step in management? Bimanual massage,
What medications (and in what order) would you use to stop the bleeding? Pitocine 80units bolus IV or 10units IM, Misoprostol 600mcg SL or 1000mcg rectally
What is a rare complication of this diagnosis that results in high fetal and maternal death?Uterine rupture
Syncope is probably up there with dizziness for one of my least favorite ED complaints. Our job as emergency physicians however is to triage those who had simple vasovagal episodes from those who may have significant morbidity and mortality if we just let them go home. The purpose of this blog post is to list several important things I try to look for apart from the regular obvious EKG findings that would have us worried such as an acute STEMI, SVT, V-tach. Below are several other EKG diagnoses to keep an eye out for. See if you can dx the patient prior to the answer below.
Case 1- 25 yo male with a history of palpitations presents after syncopal episode
EKG from Life In the Fast Lane
Diagnosis
Wolf-Parkinson-White syndrome
Pathophysiology
Presence of a congenital accessory pathway “Bundle of Kent” which predisposes a person to deadly arrythmias.
Orthodromic
Antidromic
EKG findings
Shortened PR interval < 120ms
Delta Wave: Slurred begning upstroke of the QRS complex
Wide QRS interval
Delta wave. Picture taken from wikiem.org
Management
If associated with atrioventricular reentry tachycardias we need to treat
Orthodromic conduction- Conduction from AV node back through accessory pathway
Narrow qrs
Stable Treatment- treat like SVT with Vagal maneuvers adenosine, procainamide, calcium channel blockers
Unstable treatment synchronized cardioversion
Antidromic conduction- conduction through the accessory pathway and retrograde via SA Node
Likely admission unless asymptomatic, known WPW, and cardiology can follow up very closely
Case 2: 25 yo male with fever presents after syncopal episode
EKG from Life in the Fast Lane
Diagnosis
Brugada Syndrome
Pathophysiology
Genetic sodium channelopathy with high risk for sudden cardiac death and Vfib arrest
Dx requires ECG findings as well as:
Documented ventricular fibrillation
Family history of sudden cardiac death
Similar EKG in family members
Syncope
EKG findings
Right bundle branch pattern RSR’ in leads V1, V2
ST elevation in precordial leads V1-V3
ST elevations can have different morphology: >2mm in type 2 Brugada, > 1mm in type 3 Brugada
Management and Disposition
If incidental- No acute treatment but will need very close follow up for pacemaker
If symptomatic- had syncopal episode and are now fine or active arrythmia admit with cardiology consultation for pacemaker
Case 3. 25 yo male with syncopal episode at soccer practice
Diagnosis
Hypertrophic obstructive cardiomyopathy aka HOCM
Pathophysiology
Genetic (1 in 500 individuals) condition which causes hypertrophy of cardiac muscles leading to possible left ventricular outflow tract obstruction and syncope
Decreased compliance leads to poor filling and cardiac function
Symptoms worse with exertion typically
EKG findings
Left ventricular hypertrophy criteria S wave in V1 + R wave in V5 > 35mm
“Dagger-like” Q waves can bee seen in anterolateral leads
Management
Avoid exertional activities
Needs ICD placement
Can have surgical myomectomy performed
Beta Blockers
Disposition
Cardiology consultation and admission for echo and ICD evaluation
Case 4 25 yo male with syncopal episode while watching tv recently started on azithromycin
EKG from wikiem.org
Diagnosis
Long QT syndrome (LQTS)
Pathophysiology
Group of inherited conditions resulting in delayed ventricular repolarization
EKG findings
Corrected QT interval QTc of >450 in men and > 460 in women
QTc = QT /√R-R
Can guestimate about half of the R-R interval as well
Management
Unstable- defibrillation
Stable
Stop any QT prolonging medication
Magnesium sulfate IV
Consider amiodarone
Disposition
If symptomatic or QT > 500 consider admission
Case 5. 25 yo male presents s/p sudden cardiac arrest with ROSC after defibrillation
EKG from Life in the Fast Lane
Diagnosis
Arrhythmogenic Right Ventricular dysplasia
Pathophysiology
Inherited myocardial disease where you get fibrofatty infiltration and thinning of the RV myocardium, RV dilation, global systolic dysfunction
EKG findings
Epsilon wave – small positive deflection at the end of the qrs (most specific finding, seen in 30% of patients)
Can be confused for Osborn J wave
Prolonged S wave Upstroke (95% of patients) similar to WPW
Management
Treat arrythmias
Sotalol and Amiodarone
Urgent ICD placement
Disposition
Admission if symptomatic
Very close follow up if incidental finding
Summary:
In every EKG one should take a quick glance at the wave morphology to look for signs of WPW, HOCM, ARVD, LQTS, and Brugada ( I know that’s a lot of abbreviations). I can’t say that I have found any of these yet in my syncope patients but as Dr. Thomas told me if you never look for them you will never find them.
Extra: I found a great pictoral from ALIEM for can’t miss EKG findings that has the changes listed above and more. I have listed it below for your reference.
Sources: Life in the Fast Lane , Wikiem.org, ALIEM