Ovarian Torsion, Garrett Stults D.O.:
- R > L ovary due to increased length of utero-ovarian ligament and no sigmoid colon to stabilize
- Incidence unknown, often missed, majority of cases in reproductive cases, peds cases around 15%
- Risk factors: previous torsion, ovary >4cm, 85% have ovarian mass
- Acute onset of moderate/severe pain, n/b, fever, mass, can have peritoneal signs but this should raise concern for adnexal necrosis
- CT noninferior to ultrasound, if CT is concerning then do not delay gyn consult for ultrasound
- Definitive diagnosis is made by direct visualization of ovary
- Can de-torse or may have to do oophoropexy
Hyperemesis Gravidarum, Dominic Aiello, M.D.:
- Nausea/vomiting of pregnancy: normal vitals, normal physical, normal labs, 60-80% of pregnancies in first trimester
- Hyperemesis has increased incidence in lower socioeconomic class and non-Caucasian populations
- Complications: orthostatic hypotension, electrolyte abnormalities, transaminitis, Mallory Weiss tears, Wernicke encephalopathy, increased risk of pre-e, abruption, and low birth weight if in 2nd trimester
- Treatment:
- Nonpharmacologic: avoid triggers, small meals, avoid stress, ginger, P6 acupressure wristbands
- Pharmacologic: pyridoxine, doxylamine. If persistent can add dimenhydrinate (Dramamine), Benadryl, prochlorperazine, promethazine,
- No dehydration: metoclopramide, ondansetron, promethazine, trimethobenzamide
- Dehydration: D5NS If ketonuria is present, can add in methylpred taper
Trauma in Pregnancy: the 2 for 1 plan, Melissa Platt, M.D.:
- Trauma is #1 cause of hospitalization for pregnant women (7-8% including falls)
- MVA 2/3
- Respiratory changes:
- Upper airway: mucosal edema, epistaxis, estrogen induced
- Lungs: multiple changes related to capacity, increasing RR by 2-3 breaths/min
- Oxygen consumption increased, respiratory alkalosis—pay attention if you get a gas
- Cardiovascular changes:
- Cardiac output rises 30-50%
- ½ of this occurs by 8w of pregnancy
- Influenced by posture
- Preload increased due to rise in blood volume, afterload reduced due to decline in SVR, HR increased by 15-20 BMP, EF remains unchanged (reliable indicator of LV function)
- Appears enlarged on CXR, different projection/rotation
- Apex at 4th intercostal space instead of 5th
- EKG with LAD, ST depression, 28% PVC
- BP typically falls but later returns to baseline
- Cardiac output rises 30-50%
- Placenta
- Low resistance circulation, no neuronal input
- Vascular resistance is determined more by things like endothelin, NO, not epi
- Placental flow 400-600cc/min
- Blastocyst implants in innermost uterine wall, uterine blood supply is rich
- Other changes:
- higher diaphragm
- chest becomes more barrel shaped with increased diameter
- slowed GI motility, slowed gastric emptying
- normal to have small amounts of intraperitoneal fluid
- widened symphysis pubic and sacroiliac joints
- renal changes
- plasma volume expands, peaks at 28-34 weeks
- physiologic anemia with decreased blood viscosity
- Trauma general principles:
- Focus initially on ABCs, management dictated by severity initially geared toward maternal stabilization, what’s best for mom is going to be what’s best for baby
- Do not under diagnosis or under treat secondary to unfounded fears of fetal effects
- Place on O2 early due to decrease in FRC and increased O2 consumption
- Recognize shock early
- Four factors in maternal trauma/surgery that predict fetal morbidity/mortality: hypoxia, drug effects, infection, preterm labor
- Decreased maternal hematocrit >50% or decreased MAP 20% or paO2 <60 à fetal hypoxia
- Anesthesia-surgery is best between weeks 13-23
- Secondary survey:
- Examine for non-obstetric injury, fetal heart tones, speculum exam to r/o SROM or VB
- Chest tubes need to be one intercostal space higher
- Weigh risk/harms of CT
- Shared decision making between Ob-Gyn, trauma, EP, and patient
- Most gestational ages: check fetal heart tones
- Continuous fetal monitoring is appropriate only if OB is willing to act on it (viable fetus)
- If heart tones are absent regardless of gestational age, no fetal resuscitation
- During acute phase, uterine contraction monitoring is appropriate
- Remember you cannot r/o abruption with ultrasound (50% accurate or less)
- Abruption:
- Can occur with no sign of injury externally
- Maternal mortality 1-2%, fetal 20% +
- VB, abdominal cramps, uterine tenderness, amniotic fluid leakage, change in FHT, maternal hypotension
- Labs:
- Fibrinogen/KB test
- Any patient who is Rh negative with abdominal trauma should receive Rhogam
- Utilize ultrasound, MRI as needed
- Penetrating trauma:
- Remember intraabdominal organs change position
- Electrical burns: fetus has lack of resistance to current = high fetal mortality
- Other burns: silver sulfadiazine cream- used sparingly due to risk of kernicterus
- Pelvic fractures:
- Increased risk of shock, bladder, urethra injuries
- Fetal skull fracture, fat embolism, vaginal lacerations
- Is pelvic fracture an absolute contraindication for vaginal delivery? NO, depends on severity/type and compromise of pelvic inlet
- Seat belt: lap belts alone increase abruption due to forward flexion and uterine compression, educate to wear low across pelvis
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- Randomly assigned to dates
- Format 7 cases, 15 min each, 2023 changing to 5 single patient, 2 structured interview
- Practice practice practice
- Review books, courses, online resources and practice cases
- Structured interview: intended to assess clinical judgement and though process for decision making
- Expect “why did you do xyz” question
- What are you looking for
- Interpret labs
- Initial case stimuli with brief history, vitals.
- Take note of all abnormal vitals, must be addressed
- Labs
- If certain lab is unavailable, move on
- Should not be borderline
- Occasionally may be given results you did not ask for, standardized results for all applicants, not that you missed something
- Imaging
- Usually clear, not designed to be tricky/subtle
- All static
- If unavailable, may need to stabilize or find alternative means of diagnosis
- Talk to patient as if they are present
- Ask for whatever you need
- Pharmacy, poison control, family, EMS, etc
- If someone says no or disagrees with you- you are allowed to argue
- However, if you have tried to convince and they still refuse, you may be going down wrong path
- If you receive prompting of “anything else you would give/anyone else you would call?”, pause and reconsider
- Approach: HAVE A SYSTEM, if you don’t, you will skip steps and miss important findings
- HPI: generally, answers are direct enough that you can get a thorough history quickly
- Think level 5 charting: ask med, surgical, family, social hx. Ask meds. Ask allergies.
- Exam: head to toe on all cases, keep list, take notes, practice to move through efficiently
- Give orders clearly at a speed in which your examiner can keep up
- Be aware of scoring system, look at ABEM website
- ABC survey first in unstable patients, then interventions to stabilize, then secondary survey, gather more history to supplement along with additional medical info
- ABC interventions: accucheck, bil IV, cardiac monitor (supplemental O2 PRN, cont pulse ox, BP), draw rainbow of labs, EKG, family/EMS hang around, gown/expose, “hello” introduce to patient, immobilization/isolation
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Substance Use Disorders in the ED, Richard Cales, M.D.
- Dependence- reliance on a substance to prevent withdrawal
- Easily managed with medication, can be resolved with slow taper,
- Not a unique property for many substances, but rather a normal and expected distraction from the real problem of addiction
- Addiction: unlike physical dependence, is abnormal and classified as a disease
- Primary condition associated with uncontrollable cravings, inability to control use, compulsive use, continued use despite harm to self or others.
- Currently characterized as substance use disorders
- Polysubstance use is the rule as opposed to exception for patients with severe substance use disorder (frequently nicotine and alcohol, also methamphetamines in this area)
- Always think- what else are they taking?
- Diagnosis:
- Severity groups based off of specific criteria associated with impaired control, social impairment, risky use, drug dependence
- POC urine drug testing has been used widely and remains appropriate for screening low risk populations (workplace, schools, military, etc).
- Massive number of false positives, negatives
- Should not be used for management, use as red flag to refer/obtain additional testing such as mass spectrometry
- ED options for OUD:
- New term is MOUD (medication for opioid use disorder) rather than MAT (medication assisted treatment)
- Buprenorphine slowly replacing methadone as standard of care
- Available as daily sublingual tablets/films or monthly depot injections
- Most common ED presentations:
- Overdose requiring admission- managed with Narcan, admission, eventual referral
- Overdose not requiring admission: managed with symptomatic treatment and referral
- OUD patient in withdrawal: managed with buprenorphine induction and referral
- OUD patient not in withdrawal seeking treatment: managed with referral only
- Vulnerability to addiction is 50% genetic (derived from twin studies)
- Addiction (defined as severe SUD) is chronic
- Subject to acute exacerbation, similar to severe COPD, CHF
- Requires lifelong treatment (as opposed to tapering, which is often used for mild SUD)
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